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Respiratory Medicine
Volume 104, Issue 10
, Pages 1404-1409
, October 2010
Mediators of inflammation in nasal lavage from aspirin intolerant patients after aspirin challenge
References
- . The broken balance in aspirin hypersensitivity. Eur J Pharmacol. 2006;533:145–155
- . Natural history of aspirin-induced asthma. Eur Respir J. 2000;16:432–436
- . Aspirin intolerance and allergy to house dust mites: important factors associated with development of severe asthma. Ann Allergy Asthma Immunol. 2004;92(4):453–458
- Pathogenesis, diagnosis, and treatment of aspirin intolerance. Ann Allergy Asthma Immunol. 2007;99:13–21
- . Lipoxin A4 generation is decreased in aspirin-sensitive patients in lysine-aspirin nasal challenge in vivo model. Allergy. 2009;64:1746–1752
- . Aspirin-induced asthma: advances in pathogenesis, diagnosis and management. J Allergy Clin Immunol. 2003;111:913–921
- Adhesion molecules and their ligands in nasal polyps of aspirin-hypersensitive patients. Ann Allergy Asthma Immunol. 2006 Jan;96(1):105–111
- EAACI/GA2LEN guideline: aspirin provocation tests for diagnosis of aspirin hypersensitivity. Allergy. 2007;62(10):1111–1118
- Release of peptide leukotriene into nasal secretions after local instalation of aspirin in aspirin-sensitive patients. Am Rev Respir Dis. 1992;145:65–69
- Nasal provocation test with lysine-aspirin for diagnosis of aspirin sensitive asthma. J Allergy Clin Immunol. 1998;101:581–586
- . The “nasal pool”-device for challenge and lavage of the nasal mucosa in children: histamine-induced plasma exudation responses. Pediatr Allergy Immunol. 1997;8(3):137–142
- Visual analog scales can assess the severity of rhinitis graded according to ARIA guidelines. Allergy. 2007;62(4):367–372
- Monocyte chemotactic protein-3 (MCP3) interacts with multiple leukocyte receptors. C-C CKR1, a receptor for macrophage inflammatory protein-1 alpha/Rantes, is also a functional receptor for MCP3. J Biol Chem. 1995;270:22123–22128
- RANTES activates antigen-specific cytotoxic T lymphocytes in a mitogen-like manner through cell surface aggregation. International Immunology. 2000;12(8):1173–1182
- RANTES, eotaxin and eotaxin-2 expression and production in patients with aspirin triad. Allergy. 2003;58(11):1165–1170
- Comparison of plasma eotaxin family level in aspirin-induced and aspirin-tolerant asthma patients. Chest. 2005;128(5):3127–3132
- . Eotaxin and the attraction of eosinophils to the asthmatic lung. Respir Res. 2001;2:150–156
- Kinetics of eotaxin generation and its relationship to eosinophil accumulation in allergic airways disease: analysis in a guinea pig model in vivo. J Exp Med. 1997;186:601–612
- Airway lipoxin A4 generation and lipoxin A4 receptor expression are decreased in severe asthma. Am J Respir Crit Care Med. 2008;178:574–582
- Prostaglandin E2 systemic production in patients with asthma with and without aspirin hypersensitivity. Thorax. 2008;63:27–34
- . Serum interleukin-5 in aspirin-induced asthma. Clin Exp Allergy. 2001;31(7):1036–1040
- Peripheral blood T lymphocytes from asthmatic patients are primed for enhanced expression of interleukin (Il)-4 and Il-5 mRNA: associations with lung function and serum IgE. Clin Exp Allergy. 1999;29:772–779
- Clinical significance of eosinophilic cationic protein levels in nasal secretions of patients with nasal polyposis. Eur Arch Otorhinolaryngol. 2009;266(7):981–986
- . Enhanced expression of the leukotriene C4 synthase due to overactive transcription of an allelic variant associated with aspirin-induced asthma. Am J Respir Cell Molec Biol. 2000;23:290–296
PII: S0954-6111(10)00196-4
doi: 10.1016/j.rmed.2010.04.017
© 2010 Elsevier Ltd. All rights reserved.
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Respiratory Medicine
Volume 104, Issue 10
, Pages 1404-1409
, October 2010
